In particular, the study focuses on whether the process can counteract two key indicators of biological aging: shortening of DNA telomeres and consequent accumulation of sensuous cells.
The telomere is the end of the chromosome. Telomeres are composed of repetitive sequences of non-coding DNA that serve as bumpers to protect the chromosome from damage during replication. Each time a replica occurs, these bumpers take a hit, making it shorter and shorter.
Once the telomere reaches a certain length, the cell can no longer replicate, leading to sensory cells: aging, defective cells that eventually lead to cognitive or other age-related disabilities and diseases such as cancer.
Every 20 minutes, participants were asked to remove their masks for five minutes, bringing their oxygen back to normal levels. However, during this period, the researchers found that fluctuations in free oxygen oxygen concentrations were interpreted at the cellular level as lack of oxygen – rather than interpreting the full level of oxygen oxygen.
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In other words, frequent exposure to intermittent hyperoxia (increased oxygen levels) induces many mediators and cellular mechanisms that are usually induced during hypoxia (a decrease in oxygen levels). is coming.
“The oxygen fluctuations we generate are important,” he said. Jerusalem Post. “During this process, a state of oxygen deprivation resulted, which led to cell regeneration.”
Experimental interruptions include improvements in focus, speed of information processing, and executive functions, which generally decline with age and about which more than 60% of people have expressed concern for more than 60 years.
Participants did not make any lifestyle, diet, or medication changes during the study period, which may change the results.
“We are not [just] The decline is slowing – we are moving backwards in time.
According to their work, the changes were exactly how the bodies of the participants were at the cellular level 25 years ago.
Efrati has been studying how to counteract the aging process for over a decade. He said the study was the first evidence that the cellular basis for the aging process could be reversed, adding that it “opens the way for many young scientists to hope and target aging as a reversible disease.”
It can also enable doctors and scientists to monitor the length of telomeres and find ways to develop drugs that can help them grow back when needed.
Will it make people live longer?
The duration of its impact in long-term follow-ups remains to be determined, Ifti said. But he added: “Maybe yes. We know that people with short telemarks die earlier, so it makes sense. “
Another disadvantage of the study was its limited sample size.
