From images Cloudy chest scans and shortness of breath patients are bound to the ventilator, we are conditioned to think of Covid-19 as a respiratory disease. But it’s not just about the lungs. From the earliest days of the epidemic, doctors were discovering that the novel coronavirus infection could rupture other parts of the body, including the brain, blood vessels, and heart. Early outbreak data from China, New York City and Washington state of Washington state that 20 to 30 percent of hospitalized patients in Covid-19 showed signs of cardiac injury.
These patients tend to get sick and have died more often than patients without cardiac complications without immediate alarm brick laying. After all, people with serious cases of Covid-19 were serious enough in that hospital. Most people who contract the virus experience a spectrum of less-severe symptoms. One in three never gets sick. But now there is evidence that the virus can damage the heart, even in people who have mild symptoms or none at all, especially if they exercise while getting infected.
Last month, when league commissioners of the Big Ten and Pack-12C college ledge conferences announced they would postpone the fall sports season of 2020, a major factor they said was concerns about something called myocarditis. He is a cardiologist-spokesman for what happens when the muscular walls of the heart become inflamed, weakening the limb and making it more difficult to pump blood. It is not a newly discovered condition, and it rarely changes, but when it does, it is most likely caused by an infection. Attacking viruses, bacteria, amoebas, yeasts and worms have also been shown for it.
What they have in common is that they whip up the body’s immune system in an aggressive mode and lead to inflammation. If a person is sick and rests during recovery, most of the inflammation goes away and the heart muscle heals on its own. But strenuous activity when the heart is weak can cause swelling in the legs, dizziness, difficulty breathing, and in serious cases can lead to irregular heartbeat, cardiac arrest, and sudden death.
These more extreme results are mostly seen in competitive athletes. That’s why cardiologists are urging caution about reversing the mid-game epidemic. Just last month, Florida State basketball player Michael Ojo died of heart problems while playing in a pro-league match in Serbia, shortly after the 27-year-old recovered from Kovid-19.
Leading to heart injuries in student athletes to prevent epidemics, doctors at Ohio State University have developed a new protocol, says OSU cardiologist and assistant professor of internal medicine Saurabh Rajpal. The protocol requires any athlete to obtain a clinical examination, blood test, electrocardiogram and MRI – expensive and less used imaging technology – for the diagnosis of Covid-19. Between June and August Gust, 26 men and women from the school’s football, soccer, lacrosse, basketball, L and track teams were shown recovering from Covid-19. MRI showed signs of myocarditis – inflammation of the heart muscle in four of them. Of those, Bay never experienced any of the symptoms of Covid-19. The series of cases was reported in the journal on Friday JAMA Cardiology.
Because athletes’ hearts were not conceived before their Covid-19 infection, and because they did not contract the virus – did not match the same controls as the same ones, it is impossible to say with certainty whether the damage was observed by the virus. . But Rajpal, a co-experimenter with the study, says myocarditis is caused by other viral infections, and SARS-CoV-2 is no different. “It’s important for people to know that Covid-19 can affect the heart.”
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