Extreme levels of ‘friendly fire’ antibodies in the immune system can provoke severe coronavirus symptoms and cause ‘long covid’, scientists say.
Coronavirus patients have higher levels of tontibodies in their blood that block the body’s covid-tackling antibodies and attack many areas, including the brain, blood vessels, and liver.
This may provide an account of why some covid victims experience long-term symptoms – such as fatigue, shortness of breath and brain problems.
Extreme levels of ‘friendly fire’ antibodies in the immune system can provoke severe coronavirus symptoms and cause ‘long covid’, scientists say (file image)
Researchers at Yale University calculated the number of ‘autoantibodies’ in the blood of 194 hospital staff and covid patients compared to 194 healthy staff members.
They found that non-infected people had far fewer autoantibodies than those with the virus.
The second set of tests found that people with more tontibodies – who attack the same organs that cause covid damage – experience worse Covid-19 symptoms.
Scientists fear that the negative effects of ‘friendly fire’ could continue for an extended period of time, leading to long covid.
Covid-19 is described as a short-term illness caused by a novel SARS-CoV-2 coronavirus novel infection. Public health officials say people will recover in two weeks or more.
However, it has become increasingly clear that this is not the case for everyone, and the two-week period is just the ‘acute illness’ phase.
The North Bristol NHS Trust’s Discover Project, which is studying the long-term effects of coronavirus, found that out of a total of 110 patients examined over a three-month period, most (74 per cent) had a persistent symptom after at least twelve. Weeks.
Aaron Ring, a Yale immunobiologist and senior author of the study, told The Guardian: ‘Covid-19 patients develop autoimmune diseases that actually interfere with the immune response to the virus.
‘We definitely believe that these auto tontibodies are harmful to Covid-19 patients.
‘Because antibodies can last longer, it is conceivable that they may contribute to the development of long-covid diseases.’
Antibodies are disease-fighting proteins that are stored by the immune system by hanging on their spike proteins to fight future invaders.
Tontibodies, on the other hand, have the wrong shape to bind instead of attaching proteins to human cells.
In the study, Dr Ringe – along with Akiko Iwasaki, a professor of immunobiology – conducted a study that has not yet been reviewed or published by Peer.
The news follows that the chances of Xvard University’s Covid-19 vaccine being introduced by the end of this year are ‘very high’.
The Oxford and pharmaceutical giant AstraZeneca vaccines are not yet approved for use in the UK – the Medicines and Healthcare Products Regulatory Agency (MHRA) is still reviewing trial data.
The government has secured 100 million doses of the vaccine, with the key ready for a million key workers once approved.
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But he insisted that different vaccines are used to treat different patients – using different techniques.
Professor Gilbert told the BBC’s Andrew Marsh Show: ‘It depends on the age group you are in and the JCVI (Joint Committee on Vaccination and Immunization) priorities.
Oxford University Vaccine Professor Sarah Gilbert (pictured) says the chances of getting a jaw before the end of 2020 are ‘very high’
‘I think the chances are much higher. But we need multiple vaccines, all countries need multiple vaccines, the world needs multiple vaccines and if possible, we need vaccines made using different technologies. ‘
He said these companies faced potential problems in the supply of raw materials producing doses, which could slow the vaccine rollout if other pockets were not available.
‘So multiple shots on goal, many iron in the fire, which we really need,’ he added.
Professor Gilbert’s optimistic assessment is steadily rising in the UK’s coronavirus case load – with 18,447 more coronavirus cases reported last Sunday, up from 1,175 last Sunday.
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